List of Autoimmune and Autoimmune-Related Diseases
Acute Disseminated Encephalomyelitis (ADEM)
Acute necrotizing hemorrhagic leukoencephalitis
Addison’s disease
Agammaglobulinemia
Alopecia areata
Amyloidosis
Ankylosing spondylitis
Anti-NMDA Receptor Encephalitis
Anti-GBM/Anti-TBM nephritis
Antiphospholipid syndrome (APS)
Autoimmune angioedema
Autoimmune aplastic anemia
Autoimmune dysautonomia
Autoimmune hepatitis
Autoimmune hyperlipidemia
Autoimmune immunodeficiency
Autoimmune inner ear disease (AIED)
Autoimmune myocarditis
Autoimmune pancreatitis
Autoimmune retinopathy
Autoimmune thrombocytopenic purpura (ATP)
Autoimmune thyroid disease
Autoimmune urticaria
Axonal & neuronal neuropathies
Balo disease
Behcet’s disease
Bullous pemphigoid
Cardiomyopathy
Castleman disease
Celiac disease
Chagas disease
Chronic fatigue syndrome
Chronic inflammatory demyelinating polyneuropathy (CIDP)
Chronic recurrent multifocal ostomyelitis (CRMO)
Churg-Strauss syndrome
Cicatricial pemphigoid/benign mucosal pemphigoid
Crohn’s disease
Cogans syndrome
Cold agglutinin disease
Congenital heart block
Coxsackie myocarditis
CREST disease
Essential mixed cryoglobulinemia
Demyelinating neuropathies
Dermatitis herpetiformis
Dermatomyositis
Devic’s disease (neuromyelitis optica)
Discoid lupus
Dressler’s syndrome
Endometriosis
Eosinophilic fasciitis
Erythema nodosum
Experimental allergic encephalomyelitis
Evans syndrome
Fibromyalgia
Fibrosing alveolitis
Giant cell arteritis (temporal arteritis)
Glomerulonephritis
Goodpasture’s syndrome
Granulomatosis with Polyangiitis (GPA) see Wegener’s
Graves’ disease
Guillain-Barre syndrome
Hashimoto’s encephalitis
Hashimoto’s thyroiditis
Hemolytic anemia
Henoch-Schonlein purpura
Herpes gestationis
Hypogammaglobulinemia
Idiopathic thrombocytopenic purpura (ITP)
IgA nephropathy
IgG4-related sclerosing disease
Immunoregulatory lipoproteins
Inclusion body myositis
Insulin-dependent diabetes (type1)
Interstitial cystitis
Juvenile arthritis
Juvenile diabetes
Kawasaki syndrome
Lambert-Eaton syndrome
Leukocytoclastic vasculitis
Lichen planus
Lichen sclerosus
Ligneous conjunctivitis
Linear IgA disease (LAD)
Lupus (SLE)
Lyme disease, chronic
Meniere’s disease
Microscopic polyangiitis
Mixed connective tissue disease (MCTD)
Mooren’s ulcer
Mucha-Habermann disease
Multiple sclerosis (MS)
Myasthenia gravis
Myositis
Narcolepsy
Neuromyelitis optica (Devic’s)
Neutropenia
Ocular cicatricial pemphigoid
Optic neuritis
Palindromic rheumatism
PANDAS (Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcus)
Paraneoplastic cerebellar degeneration
Paroxysmal nocturnal hemoglobinuria (PNH)
Parry Romberg syndrome
Parsonnage-Turner syndrome
Pars planitis (peripheral uveitis)
Pemphigus
Peripheral neuropathy
Perivenous encephalomyelitis
Pernicious anemia
POEMS syndrome
Polyarteritis nodosa
Type I, II, & III autoimmune polyglandular syndromes
Polymyalgia rheumatica
Polymyositis
Postmyocardial infarction syndrome
Postpericardiotomy syndrome
Progesterone dermatitis
Primary biliary cirrhosis
Primary sclerosing cholangitis
Psoriasis
Psoriatic arthritis
Idiopathic pulmonary fibrosis
Pyoderma gangrenosum
Pure red cell aplasia
Raynauds phenomenon
Reflex sympathetic dystrophy
Reiter’s syndrome
Relapsing polychondritis
Restless legs syndrome
Retroperitoneal fibrosis
Rheumatic fever
Rheumatoid arthritis
Sarcoidosis
Schmidt syndrome
Scleritis
Scleroderma
Sjogren’s syndrome
Sperm & testicular autoimmunity
Stiff person syndrome
Subacute bacterial endocarditis (SBE)
Susac’s syndrome
Sympathetic ophthalmia
Takayasu’s arteritis
Temporal arteritis/Giant cell arteritis
Thrombocytopenic purpura (TTP)
Tolosa-Hunt syndrome
Transverse myelitis
Ulcerative colitis
Undifferentiated connective tissue disease (UCTD)
Uveitis
Vasculitis
Vesiculobullous dermatosis
Vitiligo
Wegener’s granulomatosis (now termed Granulomatosis with Polyangiitis (GPA)
Diabetes (Type 1 diabetes, juvenile or autoimmune diabetes)
Idiopathic pulmonary fibrosis
Systemic sclerosis
Find The Common Thread Foundation 
I think you should add Adrenal Insufficiency. Addison’s is only one form of this disease. There’s CAH (congenital adrenal hyperplasia), Adrenal Fatigue, insuffiencies of the pituitary and hypothalamus can cause Secondary Adrenal Insuffiency, and many other conditions fall under this category. Thank you for your consideration!
Thanks Walter. Not sure who you are answering, but this is good info. I have found that many of us go through a similar experience in trying to find a diagnosis. We need to educate the medical pros as well as the general public as autoimmune is not on their radar when diagnosing problems. Now with the new trend of having doctor’s offices post signs that say “One Symptom per Visit” you know they’re are not interested in dealing with someone who has a systemic problem (ironically we’re great for their pocketbooks as we are the ultimate in return clientele.) Stay away from a doctor who has that sign up because he/she is not interested in healing, just in making money. That is not a doctor but a businessman.
Cool!! Thanks Gang, this is great. I can’t pull the photo out so maybe you can post it on our Photobucket page, the link is on the main page. Muchas Gracias!
February 13, 2013 I went through a difficult time and was diagnosed with Wegener’s Granulomatosis in April of 1999. After going through chemo, I was left with few side-effects. However, in my case, I am convinced the cause of my disease was chronic strep-throat. I was diagnosed with strep-throat at least three times and suffered from a very dry throat. I used to go throught a package of Halls cough drops a day, even then, I would sometimes lose my voice. About four years ago, a huge lump developed on my neck. My doctor suggested that a lymph node was infected and after taking a very strong antiobiotic, the swelling disappeared and my throat is no longer dry. Anonymous
I’m sorry to hear you have been dealing with WG – that’s the disease I have as well. After reading a whole lot of stories and experiences with WG, I’m thinking that what you were told was Strep Throat was probably a smouldering WG that because it was misdiagnosed was elevated to a point where it couldn’t be eliminated and is now with you for ever. I’m thinking the lump in the throat is WG related too as is the dryness in the throat. These are all symptoms that are within the perameters (not that there really is such a thing with WG) of where WG is supposed to be active, all within our respiratory system (and kidneys). I know of many other areas that get affected but that’s the main ones. I have a Wegener’s blog if you’d like to go and see some other stories and some research and a survey with over 900 Weggies that I made up last time I was in the hospital. http://www.weggiesunite.blogspot.com
All the very best to you and hope that your journey is easier now than it was in the early stages. I hope that we can find a cure for autoimmunity and by default this would give us a cure for Wegener’s… this is my goal, and I will do whatever I can to see it come to fruition.
Marta
Wonderful list! Please include Anti-NMDA Receptor Encephalitis to your list. Very rare! The medical community is grasping at straws treating patients. It is usually misdiagnosed with patients admitted to psychiatric hospitals which only delays the much needed medical treatment. The insurance companies, as well, are having difficulty in justifying the necessary treatments and very necessary therapy the patients need immediately as they return to “normal”.
Good List…Would like to see the newly identified (since 2007) Anti-NMDA Receptor Encephalitis added to this list. A huge amount of awareness raising and education are still needed, as many go undiagnosed or misdiagnosed. The results can lead to death or persons being institutionalized in psychiatric hospitals or to nursing homes. Recovery is slow and up and down. Treatment should be as quick as possible. We have 2 support groups on FB: The NMDA Antibodies support group and the Anti-NMDA Encephalitis in Canada Group.
WOW thanks for leinttg us know about this!!! We are a semi-gluten free home. Hubby can’t have the gluten and I can’t have corn so it makes it hard for us to eat out. I love their pizza and was bummed because we couldn’t go anymore. I know where we will be going next Thursday for dinner 🙂
Thanks for adding.
Hi Im Chris, Both diseases are aotiummune diseases. in aotiummune diseases the bodys own cells become overactive and start attacking either one part of the body (as is the case pretty much in these, with aotiummune hepatitis it is the liver, in Celiac it is an intolarance to foods that are strachy, sugared, etc). In other disease like Lupus, MS, and others they are more widespread and attack the entire body or more bodily functions or systems.I personally have Lupus, Sjgrens and Autoimmune Hepatitis.I Know a little about Celiac disease so I did some research, so I will let you know about how they diagnose that first. Celiac disease is a digestive disease that damages the small intestine and interferes with absorption of nutrients from food. People who have celiac disease cannot tolerate a protein called gluten, found in wheat, rye, and barley. Gluten is found mainly in foods but may also be found in products we use every day, such as stamp and envelope adhesive, medicines, and vitamins. to diagnose it they do several things. How is celiac disease diagnosed?Recognizing celiac disease can be difficult because some of its symptoms are similar to those of other diseases. In fact, sometimes celiac disease is confused with irritable bowel syndrome, iron-deficiency anemia caused by menstrual blood loss, Crohn’s disease, diverticulitis, intestinal infections, and chronic fatigue syndrome. As a result, celiac disease is commonly underdiagnosed or misdiagnosed.Recently, researchers discovered that people with celiac disease have higher than normal levels of certain autoantibodies in their blood. Antibodies are protective proteins produced by the immune system in response to substances that the body perceives to be threatening. Autoantibodies are proteins that react against the body’s own molecules or tissues. To diagnose celiac disease, physicians will usually test blood to measure levels ofImmunoglobulin A (IgA) anti-tissue transglutaminase (tTGA) IgA anti-endomysium antibodies (AEA) Before being tested, one should continue to eat a regular diet that includes foods with gluten, such as breads and pastas. If a person stops eating foods with gluten before being tested, the results may be negative for celiac disease even if celiac disease is actually present.If the tests and symptoms suggest celiac disease, the doctor will perform a small bowel biopsy. During the biopsy, the doctor removes a tiny piece of tissue from the small intestine to check for damage to the villi. To obtain the tissue sample, the doctor eases a long, thin tube called an endoscope through the mouth and stomach into the small intestine. Using instruments passed through the endoscope, the doctor then takes the sample.What is the treatment?The only treatment for celiac disease is to follow a gluten-free diet. When a person is first diagnosed with celiac disease, the doctor usually will ask the person to work with a dietitian on a gluten-free diet plan. A dietitian is a health care professional who specializes in food and nutrition. Someone with celiac disease can learn from a dietitian how to read ingredient lists and identify foods that contain gluten in order to make informed decisions at the grocery store and when eating out.For most people, following this diet will stop symptoms, heal existing intestinal damage, and prevent further damage. Improvements begin within days of starting the diet. The small intestine is usually completely healed in 3 to 6 months in children and younger adults and within 2 years for older adults. Healed means a person now has villi that can absorb nutrients from food into the bloodstream.In order to stay well, people with celiac disease must avoid gluten for the rest of their lives. Eating any gluten, no matter how small an amount, can damage the small intestine. The damage will occur in anyone with the disease, including people without noticeable symptoms. Depending on a person’s age at diagnosis, some problems will not improve, such as delayed growth and tooth discoloration.OK That is the Symptoms, diagnosis and treatment after the diagnosis. Onto The aotiummune Hepatitis, I will use personal experience and some information from a website.Autoimmune Hepatitis has nothing to do with Hepatitis A, B, or C. It is not contagious in any way shape or form. It can stand alone as it’s own aotiummune disease or can be triggered by another. Usually either Sjogren’s or Lupus (SLE). Usually Sjogren’s. It can also be called Lupoid Hepatitis.The symptoms most common are an extended painful upper abdomen, jaundice, Enlarged Liver, Itchiness, Fatigue, joint pain, skin rashes, nausea, vomiting, loss of appitite, dark urine, pale or grey stools.I remeber looking 5 months pregnant though I didnt eat.There are several steps of testing. First are abnormal basic Liver function tests AST, ALT, Billirubin, Sometimes a Positive ANA (Antinuclear Antibody, also found in other aotiummunes like Lupus, sjogrens, and others), a positive Anti-Smooth Muscle Test Or its inititls (sp) SMA. The pattern and level of these test help distinquish which disease may be going on. In addition you more than likely will have a liver biopsy. I had one. I had no problems. I had it, woke up was on side, and wanted to leave.. they wanted me to stay. I stayed for awhile. But not all people wake up as well as I do from surgery. I was a bit sore.Right now there are two mainstream treatments, I do not use either one, both do not work on me. Those treatments are Prednison (a class of medical steroids), and Imuran (an Immunosuppresent, used for transplant paitents, and other sutoimmune diseases). In the last several years, They have found that the medication first used for gallstones and galbladder disease called Actigall is very effective along with the natural herb Milk Thisle. Those are what my Rhuemotologists use on me. She swears by both. I was getting very close to a transplant list. I did need some IV chemo, but with that and those two other therapies I am in total remisson. In fact with proper care 7 out of 10 paitents will go into remisson. Many may still have mild attacks, mine come and go. Both disease are not desirable. No aotiummune disease is, but with proper care and following your doctors orders both can be controlled. I do co-own an aotiummune support group, we do a lot of Lupus, but we are a mixed bag of everything, mainly females but male and female.Good luckAny questions email me at Im good for the aotiummune Hepatitis Was this answer helpful?
Wow that’s one heck of a list. I too have an autoimmune disease, Sarcoidosis. Mine is in remission but I sure remember the days in pj’s! I will be participating and sending the Pajama DAy idea on to other communities.
Val – Jasper Alberta